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Monday, April 20, 2015
  Clinical Accuracy Spoken Here  

Asthma 2.0: Active Research

Scientific advances during the past 15 years have led to a greater understanding of the mechanisms of asthma and the development of new therapeutic approaches that can reduce illness and improve patient quality of life. However, many aspects of asthma’s pathophysiology remain unclear and are still under scientific investigation.

Prominent areas of research have included:

  • Interaction between genes and environmental factors and the role of this interaction in the development and expression of asthma
  • Phenotypic differences, which are thought to affect individual response to treatment

[Reference: Asthma 2.0, Module A, Pathophysiology]

  Excellence in Case Mgmt  

Asthma 2.0: Educate, Educate, Educate

Asthma can be challenging for patients (and their families), who need to follow complex drug regimens, prevent or control exposure to asthma triggers, detect and self-treat asthma exacerbations and communicate regularly with healthcare providers.

Patient education is a cornerstone of effective asthma management. Education can be provided during one-on-one and group educational sessions; during ED visits and hospitalizations; by pharmacists and school nurses; and during home visits by CMs, asthma educators and social workers.

[Reference: Asthma 2.0, Module D, CM Role]

  Improving Patient Care...  

Asthma 2.0: Asthma and Allergies

Atopy is the genetic predisposition to develop an IgE-mediated response to common environmental allergens. Atopy is found in 30% to 50% of the general population and frequently occurs in the absence of asthma.

The allergens that most commonly invoke an IgE-mediated response in atopic individuals are house-dust mites, animal dander, cockroach detritus, fungi and molds. Outdoor pollens pose less risk, although grass and ragweed pollen have been associated with seasonal asthma. It is widely accepted that the importance of inhalant sensitivity as a cause of asthma declines with advancing age.

[Reference: Asthma 2.0, Module A, Pathophysiology]


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